Help for low sperm count

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Your money shot may be a blank. About 15 percent of couples have trouble conceiving, and at least a third of the time the guy's sperm is at fault, according to the National Institutes of Health. So get busy: Sperm quality peaks in winter and early spring, an Israeli study found, and low-stress weekends offer your best chances of success. Ashok Agarwal, Ph.
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Low sperm count: what are the causes, symptoms and treatment?

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10 Everyday Habits That Lower Sperm Count - Men's Journal

Hershey Medical Center—highlight the most common blunders you might be making. Of course, it can also hurt your liver. How much is too much? The more you smoke, the more you lower your testosterone levels inside the testicle itself, which is where your sperm is made. Chronic opioids like oxycodone or hydrocodone, used for, say, people with chronic back pain, can decrease testosterone levels and degrade sperm production. Ultimately, this can cause male hypogonadism—the underproduction of sperm or testosterone, or both. Not quite the definition of manliness.
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Oligospermia

Sperm are male reproductive cells present in the ejaculate. A doctor can tell someone their sperm count using a test called semen analysis. The average sperm count is between 40 million and million sperm per milliliter. This number can vary between testing centers, with the above range coming from Resolve , a nonprofit fertility association. A doctor may recommend a semen analysis for many reasons, such as to test for possible underlying causes of infertility , how well the reproductive organs are working, or whether a surgical procedure for sterility was successful.
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Spermatogenesis, the complex process of male germ cell proliferation, differentiation, and maturation, is the basis of male fertility. In the seminiferous tubules of the testes, spermatozoa are constantly generated from spermatogonial stem cells through a stereotyped sequence of mitotic and meiotic divisions. The basic physiological principles, however, that control both maturation and luminal transport of the still immotile spermatozoa within the seminiferous tubules remain poorly, if at all, defined. Here, we show that coordinated contractions of smooth muscle-like testicular peritubular cells provide the propulsive force for luminal sperm transport toward the rete testis. Moreover, we identify P2 receptor-dependent purinergic signaling pathways as physiological triggers of tubular contractions both in vitro and in vivo.
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